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Congenital methemoglobinemia arises from globin mutations that stabilize iron in the ferric state (e.g., HbM Iwata [ 87HisTyr ], Table 127-3) or from mutations that impair the enzymes that reduce methemoglobin to hemoglobin (e.g., methemoglobin reductase, NADP diaphorase)

A lack of this vitamin may result in nail ridges, brittle and thinning hair, and hyperpigmentation of the skin

How do astrocytes participate in neural plasticity

Binding of filamin to the C-terminal tail of the calcitonin receptor controls recycling

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